Hypnopompia in the context of "Lucid dream"

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๐Ÿ‘‰ Hypnopompia in the context of Lucid dream

In the psychology subfield of oneirology, a lucid dream is a type of dream wherein the dreamer realizes that they are dreaming during their dream. The capacity to have and sustain lucid dreams is a trainable cognitive skill. During a lucid dream, the dreamer may gain some amount of volitional control over the dream characters, narrative, or environment, although this control of dream content is not the salient feature of lucid dreaming. An important distinction is that lucid dreaming is a distinct type of dream from other types of dreams such as prelucid dreams and vivid dreams, although prelucid dreams are a precursor to lucid dreams, and lucid dreams are often accompanied with enhanced dream vividness. Lucid dreams are also a distinct state from other lucid boundary sleep states such as lucid hypnagogia or lucid hypnopompia.

In formal psychology, lucid dreaming has been studied and reported for many years. Prominent figures from ancient to modern times have been fascinated by lucid dreams and have sought ways to better understand their causes and purpose. Many different theories have emerged as a result of scientific research on the subject. Further developments in psychological research have pointed to ways in which this form of dreaming may be utilized as a therapeutic technique.

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Hypnopompia in the context of Hypnagogia

Hypnagogia is the transitional state from wakefulness to sleep, also defined as the waning state of consciousness during the onset of sleep. Its corresponding state is hypnopompia โ€“ sleep to wakefulness. Mental phenomena that may occur during this "threshold consciousness" include hallucinations, lucid dreaming, and sleep paralysis.

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Hypnopompia in the context of Narcolepsy

Narcolepsy is a chronic neurological disorder that impairs the ability to regulate sleepโ€“wake cycles, and specifically impacts REM (rapid eye movement) sleep. The symptoms of narcolepsy include excessive daytime sleepiness (EDS), sleep-related hallucinations, sleep paralysis, disturbed nocturnal sleep (DNS), and cataplexy. People with narcolepsy typically have poor quality of sleep.

There are two recognized forms of narcolepsy, narcolepsy type 1 and type 2. Narcolepsy type 1 (NT1) can be clinically characterized by symptoms of EDS and cataplexy, and/or will have cerebrospinal fluid (CSF) orexin levels of less than 110 pg/ml. Cataplexy are transient episodes of aberrant tone, most typically loss of tone, that can be associated with strong emotion. In pediatric-onset narcolepsy, active motor phenomena are not uncommon. Cataplexy may be mistaken for syncope, tics, or seizures. Narcolepsy type 2 (NT2) does not have features of cataplexy, and CSF orexin levels are normal. Sleep-related hallucinations, also known as hypnagogic (going to sleep) and hypnopompic (on awakening), are vivid hallucinations that can be auditory, visual, or tactile and may occur independent of or in combination with an inability to move (sleep paralysis). Narcolepsy is a clinical syndrome of hypothalamic disorder, but the exact cause of narcolepsy is unknown, with potentially several causes. A leading consideration for the cause of narcolepsy type 1 is that it is an autoimmune disorder. Proposed pathophysiology as an autoimmune disease suggest antigen presentation by DQ0602 to specific CD4+ T cells resulting in CD8+ T-cell activation and consequent injury to orexin producing neurons. Familial trends of narcolepsy are suggested to be higher than previously appreciated. Familial risk of narcolepsy among first-degree relatives is high. Relative risk for narcolepsy in a first-degree relative has been reported to be 361.8. However, there is a spectrum of symptoms found in this study, from asymptomatic abnormal sleep test findings to significantly symptomatic.

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