Facial nerve in the context of Cranial nerve


Facial nerve in the context of Cranial nerve

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⭐ Core Definition: Facial nerve

The facial nerve, also known as the seventh cranial nerve, cranial nerve VII, or simply CN VII, is a cranial nerve that emerges from the pons of the brainstem, controls the muscles of facial expression, and functions in the conveyance of taste sensations from the anterior two-thirds of the tongue. The nerve typically travels from the pons through the facial canal in the temporal bone and exits the skull at the stylomastoid foramen. It arises from the brainstem from an area posterior to the cranial nerve VI (abducens nerve) and anterior to cranial nerve VIII (vestibulocochlear nerve).

The facial nerve also supplies preganglionic parasympathetic fibers to several head and neck ganglia.

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Facial nerve in the context of Metencephalon

The metencephalon is the embryonic part of the hindbrain that differentiates into the pons and the cerebellum. It contains a portion of the fourth ventricle and the trigeminal nerve (CN V), abducens nerve (CN VI), facial nerve (CN VII), and a portion of the vestibulocochlear nerve (CN VIII).

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Facial nerve in the context of Secretomotor

The adjective secretomotor refers to the capacity of a structure (often a nerve) to induce a gland to secrete a substance (usually mucus or serous fluid).

Secretomotor nerve endings are frequently contrasted with sensory neuron endings and motor nerve endings. An example of secretomotor activity can be seen with the lacrimal gland, which secretes the aqueous layer of the tear film. The lacrimal branch of the ophthalmic nerve (itself a branch of trigeminal nerve V1) supplies secretomotor innervation to the lacrimal gland, stimulating its secretion of the aqueous layer. However, these nerves fibers originate from the facial nerve (VII) and only travel briefly with fibers from the trigeminal nerve.

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Facial nerve in the context of Lacrimal apparatus

The lacrimal apparatus is the physiological system containing the orbital structures for tear production and drainage.
It consists of:


The blood supply to the lacrimal gland is provided by the ophthalmic artery with its branch - the lacrimal artery, while the venous blood is drained from this region via the superior ophthalmic vein. The lacrimal system is made up of a secretory system, which produces tears, and an excretory system, which drains the tears. The lacrimal gland is primarily responsible for producing emotional or reflexive tears. As tears are produced, some fluid evaporates between blinks, and some is drained through the lacrimal punctum. The tears that are drained through the punctum will eventually be drained through the nose. Any excess fluid that did not go into the punctum will fall over the eyelid, which produces tears that are cried.

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Facial nerve in the context of Mixed nerve

A mixed nerve is any nerve that contains both sensory (afferent) and motor (efferent) nerve fibers. All 31 pairs of spinal nerves are mixed nerves. Four of the twelve cranial nervesV, VII, IX and X are mixed nerves.

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Facial nerve in the context of Central facial palsy

Central facial palsy (colloquially referred to as central seven) is a symptom or finding characterized by paralysis or paresis of the lower half of one side of the face. It usually results from damage to upper motor neurons of the facial nerve.

The facial motor nucleus has dorsal and ventral divisions that contain lower motor neurons supplying the muscles of the upper and lower face, respectively. The dorsal division receives bilateral upper motor neuron input (i.e. from both sides of the brain) while the ventral division receives only contralateral input (i.e. from the opposite side of the brain).

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Facial nerve in the context of Ramsay Hunt syndrome type 2

Ramsay Hunt syndrome type 2, commonly referred to simply as Ramsay Hunt syndrome (RHS) and also known as herpes zoster oticus, is inflammation of the geniculate ganglion of the facial nerve as a late consequence of varicella zoster virus (VZV). In regard to the frequency, less than 1% of varicella zoster infections involve the facial nerve and result in RHS. It is traditionally defined as a triad of ipsilateral facial paralysis, otalgia, and vesicles close to the ear and auditory canal. Due to its proximity to the vestibulocochlear nerve, the virus can spread and cause hearing loss, tinnitus (hearing noises that are not caused by outside sounds), and vertigo. It is common for diagnoses to be overlooked or delayed, which can raise the likelihood of long-term consequences. It is more complicated than Bell's palsy. Therapy aims to shorten its overall length, while also providing pain relief and averting any consequences.

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Facial nerve in the context of Facial muscles

The facial muscles are a group of striated skeletal muscles supplied by the facial nerve (cranial nerve VII) that, among other things, control facial expression. These muscles are also called mimetic muscles. They are only found in mammals, although they derive from neural crest cells found in all vertebrates. They are the only muscles that attach to the dermis.

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Facial nerve in the context of Special visceral afferent

Special visceral afferent fibers (SVA) are afferent fibers that develop in association with the gastrointestinal tract. They carry the special sense of taste (gustation). The cranial nerves containing SVA fibers are the facial nerve (VII), the glossopharyngeal nerve (IX), and the vagus nerve (X). The facial nerve receives taste from the anterior 2/3 of the tongue; the glossopharyngeal from the posterior 1/3, and the vagus nerve from the epiglottis. The sensory processes, using their primary cell bodies from the inferior ganglion, send projections to the medulla, from which they travel in the tractus solitarius, later terminating at the rostral nucleus solitarius.

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Facial nerve in the context of Solitary nucleus

The solitary nucleus (SN) (nucleus of the solitary tract, nucleus solitarius, or nucleus tractus solitarii) is a series of neurons whose cell bodies form a roughly vertical column of grey matter in the medulla oblongata of the brainstem. Their axons form the bulk of the enclosed solitary tract. The solitary nucleus can be divided into different parts including dorsomedial, dorsolateral, and ventrolateral subnuclei.

The solitary nucleus receives general visceral and special visceral inputs from the facial nerve (CN VII), glossopharyngeal nerve (CN IX) and vagus nerve (CN X); it receives and relays stimuli related to taste and visceral sensation. It sends outputs to various parts of the brain, such as the hypothalamus, thalamus, and reticular formation, forming circuits that contribute to autonomic regulation.

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Facial nerve in the context of Geniculum

A geniculum is a small genu, or angular knee-like structure. The term is often used in anatomical nomenclature to designate a sharp knee-like bend in a small structure or organ.

For example, in the facial canal, the genicular ganglion is situated on the geniculum of the facial nerve, the point where the nerve changes its direction.

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Facial nerve in the context of Lacrimal nucleus

The salivatory nuclei are two general visceral efferent nuclei located in the caudal pons, dorsal and lateral to the facial nucleus. Their neurons give rise to preganglionic parasympathetic nerve fibers in the control of salivation. The superior salivatory nucleus supplies fibers to the intermediate nerve (part of the facial nerve (CN VII). The inferior salivatory nucleus supplies fibers to the glossopharyngeal nerve (CN IX). The nuclei may also be involved in parasympathetic control of (extracranial and intracranial) head vasculature.

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Facial nerve in the context of Dysarthria

Dysarthria is a speech sound disorder resulting from neurological injury of the motor component of the motor–speech system and is characterized by poor articulation of phonemes. It is a condition in which problems effectively occur with the muscles that help produce speech, often making it very difficult to pronounce words. It is unrelated to problems with understanding language (that is, dysphasia or aphasia), although a person can have both. Any of the speech subsystems (respiration, phonation, resonance, prosody, and articulation) can be affected, leading to impairments in intelligibility, audibility, naturalness, and efficiency of vocal communication. Dysarthria that has progressed to a total loss of speech is referred to as anarthria. The term dysarthria was formed from the Greek components dys- "dysfunctional, impaired" and arthr- "joint, vocal articulation".

Neurological injury due to damage in the central or peripheral nervous system may result in weakness, paralysis, or a lack of coordination of the motor–speech system, producing dysarthria. These effects in turn hinder control over the tongue, throat, lips or lungs; for example, swallowing problems (dysphagia) are also often present in those with dysarthria. Cranial nerves that control the muscles relevant to dysarthria include the trigeminal nerve's motor branch (V), the facial nerve (VII), the glossopharyngeal nerve (IX), the vagus nerve (X), and the hypoglossal nerve (XII).

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Facial nerve in the context of Facial motor nucleus

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Facial nerve in the context of Facial paralysis

Facial nerve paralysis is a common problem that involves the paralysis of any structures innervated by the facial nerve. The pathway of the facial nerve is long and relatively convoluted, so there are a number of causes that may result in facial nerve paralysis. The most common is Bell's palsy, a disease of unknown cause that may only be diagnosed by exclusion of identifiable serious causes.

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